MIP-1 alpha/CCL3 cDNA ORF Clone in Cloning Vector, Mouse

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MIP-1 alpha/CCL3 cDNA ORF Clone in Cloning Vector, Mouse: General Information

Gene
Species
Mouse
NCBI Ref Seq
RefSeq ORF Size
279 bp
Sequence Description
Identical with the Gene Bank Ref. ID sequence.
Description
Full length Clone DNA of Mouse chemokine (C-C motif) ligand 3.
Plasmid
Vector
Sequencing Primers
SP6 and T7 or M13-47 and RV-M
Quality Control
The plasmid is confirmed by full-length sequencing.
Screening
Antibiotic in E.coli
Ampicillin
Storage & Shipping
Shipping
Each tube contains lyophilized plasmid.
Storage
The lyophilized plasmid can be stored at ambient temperature for three months.

MIP-1 alpha/CCL3 cDNA ORF Neucleotide Sequence and Amino Acid Sequence Information

**Sino Biological guarantees 100% sequence accuracy of all synthetic DNA constructs we deliver, but we do not guarantee protein expression in your experimental system. Protein expression is influenced by many factors that may vary between experiments or laboratories.**

MIP-1 alpha/CCL3 cDNA ORF Clone in Cloning Vector, Mouse: Synonyms

AI323804 cDNA ORF Clone, Mouse; G0S19-1 cDNA ORF Clone, Mouse; LD78alpha cDNA ORF Clone, Mouse; MIP-1alpha cDNA ORF Clone, Mouse; MIP1-(a) cDNA ORF Clone, Mouse; MIP1-alpha cDNA ORF Clone, Mouse; Mip1a cDNA ORF Clone, Mouse; Scya3 cDNA ORF Clone, Mouse

MIP-1 alpha/CCL3 Background Information

CCL3 is a cytokine belonging to the CC chemokine family. Chemokines are a family of structurally related leukocyte chemoattractant cytokines that play a central role during immunoregulatory and inflammation processes. All chemokines contain four conserved cysteines linked by disulfide bonds, and two major subfamilies, namely CXC and CC, are defined on the basis of the first two cysteines which are separated by one amino acid or are adjacent. CCL3 is involved in the acute inflammatory state in the recruitment and activation of polymorphonuclear leukocytes.
Full Name
chemokine (C-C motif) ligand 3
Research Areas
References
  • Zhao RY, et al. (2005) Viral infections and cell cycle G2/M regulation. Cell Res. 15(3):143-9. Joseph AM, et al. (2005) Nef: "necessary and enforcing factor" in HIV infection. Curr HIV Res. 3(1):87-94. Muthumani K, et al. (2004) HIV-1 Vpr and anti-inflammatory activity. DNA Cell Biol. 23(4): 239-47.
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