AKT1 (Protein|Antibody|cDNA Clone|ELISA Kit)

All AKT1 reagents are produced in house and quality controlled, including 1 AKT1 Antibody, 43 AKT1 Gene, 1 AKT1 IPKit, 1 AKT1 Lysate, 1 AKT1 Protein, 2 AKT1 qPCR. All AKT1 reagents are ready to use.

Recombinant AKT1 proteins are expressed by Baculovirus-Insect Cells with fusion tags as C-His.

AKT1 antibodies are validated with different applications, which are WB, ELISA, ICC/IF, IF, IP.

AKT1 cDNA clones are full length sequence confirmed and expression validated. There are 13 kinds of tags for each AKT1 of different species, especially GFP tag, OFP tag, FLAG tag and so on. There are three kinds of vectors for choice, cloning vector, expression vector and lentivrial expression vector.

AKT1 Protein (1)


AKT1 Protein, Human, Recombinant (His Tag)


Expression host: Baculovirus-Insect Cells

Human AKT1 Protein 9097

AKT1 Antibody (1)

Application Clonality

Anti-AKT1 Antibody


Specificity: Human

Application: WB,ELISA,ICC/IF,IF,IP

Clonality: PAb

Human AKT1 Immunofluorescence(IF) 3086

AKT1 cDNA Clone (43)


AKT1 qPCR Primer (2)

AKT1 IP Kit (1)

AKT1 Lysate (1)

v-akt murine thymoma viral oncogene homolog 1 (AKT1), or protein kinase B-alpha (PKB-ALPHA) is a serine-threonine protein kinase, belonging to the Protein Kinase Superfamily. AKT1 is a major mediator of the responses to insulin, insulin-like growth factor 1 (IGF1), and glucose. AKT1 also plays a key role in the regulation of both muscle cell hypertrophy and atrophy. AKT1 activity is required for physiologic cardiac growth in response to IGF1 stimulation or exercise training. In contrast, AKT1 activity was found to antagonize pathologic cardiac growth that occurs in response to endothelin 1 stimulation or pressure overload. AKT1 selectively promotes physiological cardiac growth while AKT2 selectively promotes insulin-stimulated cardiac glucose metabolism. AKT1 deletion prevented tumor initiation as well as tumor progression, coincident with decreased Akt signaling in tumor tissues. AKT1 is the primary Akt isoform activated by mutant K-ras in lung tumors, and that AKT3 may oppose AKT1 in lung tumorigenesis and lung tumor progression. A number of separate studies have implicated AKT1 as an inhibitor of breast epithelial cell motility and invasion. AKT1 may have a dual role in tumorigenesis, acting not only pro-oncogenically by suppressing apoptosis but also anti-oncogenically by suppressing invasion and metastasis.Immune Checkpoint   Immunotherapy   Cancer Immunotherapy   Targeted Therapy